Sem Hop. Sep 22;32(56) [Edema of Quincke from the point of view of an allergy]. [Article in French]. BRUUN E. PMID: ; [Indexed for. Acquired Angioneurotic Edema (AAE) is characterized by profound swelling of the dermis associated with abdominal pain. Edema is usually painless. Images in Clinical Medicine from The New England Journal of Medicine — Uvular Angioedema (Quincke’s Disease).

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Cinnarizine may also be useful because it blocks the activation of C4 and can be used in patients with liver disease, whereas androgens cannot.

The Journal of Allergy and Clinical Immunology. It can also occur as a side effect to certain medications, particularly ACE inhibitors. We report a case of uvular edema secondary to food allergy, treated successfully with steroids.

There are as many as 80, toemergency department ED visits for angioedema annually, and it ranks as the top allergic disorder resulting in hospitalization in the Edeka. Electric shock Drowning Lightning injuries.

Mast cell tryptase levels may be elevated if the attack was due to an acute allergic anaphylactic reaction. Sign qincke to download free article PDFs Sign in to access your subscriptions Sign in to your personal account. Annals of Internal Medicine.


The acquired form is often associated with B-cell lymphoproliferative disorders. You can also find results for a single author or contributor. Chronic cases require steroid therapy, which generally leads to a good response.

If the episode occurs at all after the consumption of these foods, its onset may be delayed overnight or by some hours, making the correlation with their consumption somewhat difficult. Elevated Hyperthermia Heat syncope. Hypothermia Immersion foot syndromes Trench foot Tropical immersion foot Warm water immersion foot Chilblains Frostbite Aerosol burn Cold intolerance Erema Erythrocyanosis crurum.

Quincke’s edema, revisited.

Get free access to newly published articles Create a personal account or sign in to: Please enter User Name. Various mechanisms that interfere with bradykinin production or degradation can lead to angioedema. Current Opinion in Allergy and Clinical Immunology. Clots Thrombus Thrombosis Renal vein thrombosis.

It is particularly difficult to diagnose HAE in patients whose episodes are confined to the gastrointestinal tract. The Mount Sinai journal of medicine, New York.

Consequences of external causes T66—T78— When the patient has been stabilized, particular investigations may clarify the exact cause; complement levels, especially depletion of complement factors 2 and 4, may indicate deficiency of C1-inhibitor. Clinical mildly or nonpruritic, relapsing edema that does not respond to antiallergic treatment.


This acquired angioedema is associated with the development of lymphoma. Lymphoid and complement immunodeficiency D80—D85 Angioedema following use of angiotensin-converting enzyme ACE inhibitors is found in 1 to 2 cases per persons.

An important clue is the failure of hereditary angioedema to respond to antihistamines or steroidsa characteristic that distinguishes it from allergic reactions.

Area of swelling [1].


Create a personal account to register for email alerts with links to free full-text articles. Bruno Bissonnette, et al. Retrieved from ” https: Edemz allergic angioedema, avoidance of quncke allergen and use of antihistamines may prevent future attacks. Edema of the gastrointestinal mucosa typically leads to severe abdominal pain; in the upper respiratory tract, it can be life-threatening. Bradykinin plays a critical role in all forms of hereditary angioedema.

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